SARS-CoV-2 spike protein interacts with and activates TLR41

Biomedicines, Free Full-Text

SARS-CoV-2 spike protein induces inflammation via TLR2-dependent activation of the NF-κB pathway

SARS-CoV-2 spike protein induces a differential monocyte activation that may contribute to age bias in COVID-19 severity

Innate immune recognition against SARS-CoV-2, Inflammation and Regeneration

The battle between host and SARS-CoV-2: Innate immunity and viral evasion strategies - ScienceDirect

Frontiers Multifaceted role of SARS-CoV-2 structural proteins in lung injury

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Frontiers Mechanisms of TLR4-Mediated Autophagy and Nitroxidative Stress

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SARS-CoV-2 suppresses TLR4-induced immunity by dendritic cells via C-type lectin receptor DC-SIGN

SARS-CoV-2 spike protein induces the cytokine release syndrome by stimulating T cells to produce more IL-2

Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice

SARS-CoV-2 spike protein induces the cytokine release syndrome by stimulating T cells to produce more IL-2

SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells